Browsing by Author "Larrondo, Jorge"

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    Do not forget to check the scalp in systemic light-chain amyloidosis
    (2021) Larrondo, Jorge; Gosch, Marianne; Pena, Camila; Cabrera, Raúl; Jeraldo, Cecilia; Castro, Alex; McMichael, Amy
    Systemic light chain amyloidosis (AL) is a nonproliferative plasma cell disorder in which fragments of immunoglobulin deposit in tissues. Clinical manifestations are often nonspecific, making diagnosis challenging.Biopsy of involved organs is the gold standard to confirm the diagnosis; however, internal organ biopsy may beunsafe because of a significant risk of bleeding.1 New techniques to improve diagnosis are needed.
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    Eruptive Facial Postinflammatory Lentigo: Clinical and Dermatoscopic Features
    (Lippincott Williams & Wilkins, 2016) Cabrera, Raul; Puig, Susana; Larrondo, Jorge; Castro, Alex; Valenzuela, Karen; Sabatini, Natalia
    The face has not been considered a common site of fixed drug eruption, and the authors lack dermatoscopic studies of this condition on the subject. The authors sought to characterize clinical and dermatoscopic features of 8 cases of an eruptive facial postinflammatory lentigo. The authors conducted a retrospective review of 8 cases with similar clinical and dermatoscopic findings seen from 2 medical centers in 2 countries during 2010-2014. A total of 8 patients (2 males and 6 females) with ages that ranged from 34 to 62 years (mean: 48) presented an abrupt onset of a single facial brown-pink macule, generally asymmetrical, with an average size of 1.9 cm. after ingestion of a nonsteroidal antiinflammatory drugs that lasted for several months. Dermatoscopy mainly showed a pseudonetwork or uniform areas of brown pigmentation, brown or blue-gray dots, red dots and/or telangiectatic vessels. In the epidermis, histopathology showed a mild hydropic degeneration and focal melanin hyperpigmentation. Melanin can be found freely in the dermis or laden in macrophages along with a mild perivascular mononuclear infiltrate. The authors describe eruptive facial postinflammatory lentigo as a new variant of a fixed drug eruption on the face.
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    Frequency of the Types of Alopecia at Twenty-Two Specialist Hair Clinics: A Multicenter Study
    (2019) Vañó, Sergio; Saceda, David; Blume, Ulrike; Cucchía, José; Dlova, Ncoza; Reis, María; Grimalt, Ramón; Guzmán, Daniela; Harries, Matthew; Hoi, Anthony; Holmes, Susan; Larrondo, Jorge; Mosam, Anisa; Oliveira, Rui; Pinto, Giselle; Piraccin, Bianca; Pirmez, Rodrigo; De la Rosa, Daniel; Rudnicka, Lidia; Shapiro, Jerry; Sinclair, Rodney; Tosti, Antonella; Trüeb, Ralph; Vogt, Annika; Miteva, Mariya
    Background: The frequency of different types of alopecia is not clearly reported in recent studies. Objective: To analyze the frequency of the types of alopecia in patients consulting at specialist hair clinics (SHC) and to assess for global variations. Methods: Multicenter retrospective study including data from patients evaluated at referral SHC in Europe, America, Africa and Australia. Results: A total of 2,835 patients (72.7% females and 27.3% males) with 3,133 diagnoses of alopecia were included (73% were non-cicatricial and 27% were cicatricial alopecias). In all, 57 different types of alopecia were characterized. The most frequent type was androgenetic alopecia (AGA) (37.7%), followed by alopecia areata (AA) (18.2%), telogen effluvium (TE) (11.3%), frontal fibrosing alopecia (FFA) (10.8%), lichen planopilaris (LPP) (7.6%), folliculitis decalvans (FD) (2.8%), discoid lupus (1.9%) and fibrosing alopecia in a pattern distribution (FAPD) (1.8%). There was a male predominance in patients with acne keloidalis nuchae, dissecting cellulitis and FD, and female predominance in traction alopecia, central centrifugal cicatricial alopecia, FFA, TE, FAPD and LPP. Conclusion: AGA followed by AA and TE were the most frequent cause of non-cicatricial alopecia, while FFA was the most frequent cause of cicatricial alopecia in all studied geographical areas.
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    Lupus Miliaris Disseminatus Faciei
    (2022) Gosch, Marianne; Larrondo, Jorge
    A woman in her 40s presented with a year history of an asymptomatic, expanding papular eruption on the face. She denied any systemic symptoms. Physical examination revealed multiple small, symmetrical, reddish brown papules on her cheeks, nose, and eyelids (Figure). Skin biopsy results showed multiple perifollicular epithelioid cell granulomas, with central areas of necrosis. Staining for fungi and acid-fast bacilli yielded negative results. A computed tomography scan of the chest, complete blood cell count, and metabolic panel were normal. A diagnosis of Lupus miliaris disseminatus faciei (LMDF) was made. The patient was refractory to treatment with systemic corticosteroids, doxycycline, and oral isotretinoin. Eight months of treatment with oral dapsone (100 mg/day) produced substantial clinical improvement that was maintained at 6-month and 1-year follow-up visits
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    Trichobacteriosis axillaris caused by Dermabacter hominis
    (John Wiley & Sons, 2017) Larrondo, Jorge; Porte, Lorena; Gosch, Marianne; Cabrera, Raul; Weitzel, Thomas
    Trichobacteriosis is a frequent superficial bacterial infection of the hair, mainly affecting sweat gland bearing areas such as axillae and genital region, and also rarely the scalp.(1-3) The disease is characterized by bacterial overgrowth forming nodular concretions, usually of yellow color (infrequently black or reddish), which are firmly attached to the hair shaft.(1,4) Clinical hallmarks are rancid, acidic odor, unpleasant "dirty" sensation, and staining of clothes. Although most cases have been associated to different species of corynebacteria, the full etiologic spectrum of this infection is uncertain .(1,5) Here we report the first case of trichobacteriosis caused by Dermabacter hominis.

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